By Zeev Vlodaver MD, John R. Lesser (auth.), Zeev Vlodaver, Robert F. Wilson, Daniel J. Garry (eds.)
Coronary center illness: medical, Pathological, Imaging, and Molecular Profiles offers a accomplished photo of ischemic center ailment for practitioners, scholars, and investigators facing the numerous elements of this complicated topic. person chapters introduce the anatomy of the coronary blood vessels and cardiac improvement, whereas others give some thought to present imaging modalities applied for ischemic center illness, together with pressure echo, nuclear diagnostic exams, non-invasive coronary artery imaging, and coronary angiography. Imaging chapters offer key scientific info on thoughts and symptoms, and comprise examples of either general and irregular styles.
The precept thrust of the booklet issues coronary atherosclerosis, the pathology of that's offered together with the result of anatomic, non-invasive imaging and angiographic reviews. comparable chapters hide atherogenesis, featuring new insights into the pathophysiology of the susceptible plaque, the position of progenitor cells in vascular harm, irritation and atherogenesis, and the genomics of vascular home improvement. extra subject matters coated comprise angina pectoris, acute coronary syndromes, healed myocardial infarction and congestive center failure, catheter-based and surgical revascularization, and surgical procedure of myocardial infarction and its sequelae.
With contributions from a various staff of internationally-known physicians with large adventure within the analysis and remedy of heart illness, this publication could be a priceless source for practitioners in scientific cardiology, thoracic surgical procedure, pathology, and cardiovascular molecular examine, in addition to for college students in training.
Read Online or Download Coronary Heart Disease: Clinical, Pathological, Imaging, and Molecular Profiles PDF
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Extra info for Coronary Heart Disease: Clinical, Pathological, Imaging, and Molecular Profiles
3 Differentiation of mesodermal progenitor cells and multipotent cardiac progenitor cells (MCPCs) from pluripotent stem cells requires the expression of the Brachyury (T), Mesp1, and Mesp2 genes. Precursor cells can be differentiated by the markers Flk-1+ and Pdgfr −. Cardiac precursor cells are positive for both of these markers, while hematologic progenitor cells are only positive for Flk-1+. MCPCs can differentiate into smooth muscle, epicardial cells, endothelial cells, cardiac muscle, or cardiac conduction tissue with the appropriate environment and molecular cues The first reported molecular asymmetry in the heart is the left-sided expression of the transcription factor, Pitx2, which is required for normal heart morphogenesis [53, 54].
Stabilization of the ductus arteriosus with PGE1 is necessary for survival.
Lohr et al. (FISH) analyses [85–87]. These patients are at increased risk for poor perioperative outcomes, increased need for reoperation, and long-term complications. Infants with trisomy 21 (Down syndrome) account for a large proportion of these patients and have a 40–50% risk of CHD that most commonly includes VSD or atrioventricular canal (AVC) defect, although cyanotic CHD including TOF does occur in this patient population. Other genetic syndromes associated with a high incidence of CHD include DiGeorge syndrome (22q11 deletion syndrome), which occurs in approximately 1:4,000–1:7,000 live births [86, 88].